By William O., IV, DO Tatum, Peter W., Dr., MD Kaplan, MD Jallon Pierre
Epilepsy A-Z is a distinct compendium that offers a bullet of epilepsy-related info that's excellent for fast and simple reference. it truly is crucial for busy practitioners who wish the essence of phrases and issues appropriate to sufferer care and seizures. Physicians can fast locate solutions to medical questions, entry very important drug details, or comprehend the latest remedies and strategies for coping with the main advanced epilepsy sufferers with this concise but entire source. Key issues are summarized with present references and acceptable portraits to provide the reader a right away and direct presentation that makes A-Z a must have for the busy clinician. specified gains of this completely revised and considerably multiplied version contain:
- present in addition to condensed entire dialogue in all components of scientific epilepsy
- A to Z dictionary structure -provides a short technique to arrive on the terminology or subject of curiosity; correct
- Pearls and summaries during the e-book- demystify and make clear the place complete textbooks can t
- more recent thoughts whole with pics- help clinicians caring for even the main advanced epilepsy sufferers
- Key references summarized- supply influence for the reader and easy accessibility to comparable articles
Read or Download Epilepsy A to Z: a concise encyclopedia PDF
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Extra info for Epilepsy A to Z: a concise encyclopedia
14 ALPERS’ DISEASE ALPERS’ DISEASE Alpers’ disease is a syndrome that reflects a genetically determined disorder composed of rapidly progressive encephalopathy with intractable seizures associated with diffuse neuronal degeneration of uncertain etiology. It was first described as a progressive form of infantile diffuse neuronal degeneration. The onset is usually before the age of 1 year in the absence of a toxic, metabolic, or other structural cause. The syndrome is characterized by progressive hypotonia, cognitive deterioration, and respiratory problems; a subgroup with hepatic insufficiency is recognized.
Additionally, a unique EEG pattern of patients in coma following hypoxic/anoxic insult may also include a widespread, unreactive alpha activity that portends a poor prognosis (alpha coma). Comatose survivors are often encountered in the hospital setting with seizures occurring after resuscitation from hypoxic-ischemic injury. While the underlying insult appears to be most reflective of prognosis, status myoclonus has a poor prognosis after global hypoxic-ischemic injury [1,2]. In neonates, hypoxic-ischemic insult is the most common cause of seizures that occur in the ANTIEPILEPTIC DRUG (AED) LEVELS 23 first 48 hours of life, and isolated seizures may be followed by clusters of seizures or status epilepticus in the newborn.
The degree of retrograde and anterograde manesia is variable, and postictally a form of Todd’s phenomenon may affect the mesial temporal structures in TLE to create amnesia that may persist and mimic other disorders with memory loss such as dementia. Memory loss is a concern following temporal lobectomy for intractable epilepsy, although amnesia is rare when Wada testing demonstrates bilateral memory function. , a patient who underwent bilateral temporal lobectomy for intractable epilepsy, developed amnesia as a result, underscoring the importance of memory in those undergoing resective temporal lobe surgery.
Epilepsy A to Z: a concise encyclopedia by William O., IV, DO Tatum, Peter W., Dr., MD Kaplan, MD Jallon Pierre