Download e-book for kindle: Breast Cancer: American Cancer Society Atlas of Clinical by David P. Winchester

By David P. Winchester

ISBN-10: 1550092030

ISBN-13: 9781550092035

A part of the yankee melanoma Society's acclaimed Atlas of scientific Oncology sequence, this quantity bargains a professional evaluate of breast melanoma. issues diversity from epidemiology and genetics to prognosis, administration and reconstruction. Post-treatment care, in addition to male breast melanoma, is additionally mentioned.

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Additional resources for Breast Cancer: American Cancer Society Atlas of Clinical Oncology

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Chu KC, Tarone RE, Kessler LG, et al. S. breast cancer incidence, survival, Breast Cancer Risk and Management: Chemoprevention, Surgery, and Surveillance 2. 3. 4. 5. 6. 7. 8. 9. 10. 11. 12. 13. 14. 15. 16. and mortality rates. J Natl Cancer Inst 1996; 88:1571–9. Vogel VG. Subjects and recruitment strategies for a short-term phase II chemoprevention trial of breast cancer using surrogate endpoint biomarkers. J Cell Biochem 1993;17G:257–8. Page DL, Dupont WD, Rogers LW. Atypical hyperplastic lesions of the female breast.

117 These important differences may possibly be related to fat intake and total calories in the diet. 119,120 31 Heterocyclic amines, a group of mutagenic compounds identified in cooked foods, seem to be related to the increased risk of breast cancer associated with high intake of well-done meat. 123 The role of alcohol consumption and smoking are also being extensively investigated as possible risk factors for breast cancer. 126–129 The effects of limonene and limonene-related monoterpenes, perillyl alcohol and perillic acid, on cell growth, cell cycle 32 BREAST CANCER progression, and expression of cyclin D1 has been investigated in T-4D, MCF-7, MDA-MB231 breast cancer cell lines.

The tumors detected in users of OCs were also found to be of earlier stage than those that were detected in women that did not use OCs. Two theoretical mechanisms could explain these findings. One explanation is related to the concept of estrogen acting as a promoter rather than as a cause of the neoplastic process. Under this circumstance, it would be expected that more tumors would be detected during and following use of OCs because the estrogen content would merely be expediting the clinical appearance of a pre-existing but previously occult tumor.

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Breast Cancer: American Cancer Society Atlas of Clinical Oncology by David P. Winchester

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